By Dr. L. F. Agnati, O. Franzen, S. Ferré, G. Leo (auth.), Dr. R. Horowski, Prof. Y. Mizuno, Prof. C. W. Olanow, Prof. W. H. Poewe, Prof. P. Riederer, Dr. J. A. Stoessl, Prof. M. B. H. Youdim (eds.)
• attainable position of intramembrane receptor-receptor interactions in reminiscence and studying through formation of long-lived heteromeric complexes: concentrate on motor studying within the basal ganglia (L.F. Agnati, O. Franzen, S. Ferré, G. Leo, R. Franco, okay. Fuxe) •Self-tolerance within the immune privileged CNS: classes from the entorhinal cortex lesion version (E. Kwidzinski, L. ok. Mutlu, A. D. Kovac, J. Bunse, J. Goldmann, J. Mahlo, O. Aktas, F. Zipp, T. Kamradt, R. Nitsch, I. Bechmann) •Progressive neurodegeneration in Drosophila: a version procedure (J.-A. Tschäpe, A. Bettencourt da Cruz, D. Kretzschmar) •Effect of complicated glycation endproducts on mobile cycle and their relevance for Alzheimer`s affliction (G. Münch, J. Gasic-Milenkovic, T. Arendt) •What have we learnt from cDNA microarray gene expression stories in regards to the position of iron in MPTP brought on neurodegeneration and Parkinson`s affliction? (M. B. H. Youdim) •Animal versions of Parkinson`s affliction in rodents triggered through pollutants: an replace (E. C. Hirsch, G. Höglinger, E. Rousselet, T. Breidert, okay. Parain, J. Feger, M. Ruberg, A. Prigent, C. Cohen-Salmon, J.-M. Launay) •General features of neurodegeneration (K. A. Jellinger) •The neuromelanin of human substantia nigra: constitution, synthesis and molecular behaviour (L. Zecca, F. A. Zucca, P. Costi, D. Tampellini, A. Gatti, M. Gerlach, P. Riederer, R. G. Fariello, S. Ito, M. Gallorini, D. Sulzer) •New thoughts and instruments in imaging for the research of neurodegenerative illness (A. J. Stoessl, C. S. Lee, R. de l. a. Fuente-Fernandez) •Arguments for using dopamine receptor agonists in scientific and preclinical Parkinson`s sickness (M. Gerlach, ok. Double, H. Reichmann, P. Riederer) •The position of acetylcholine and dopamine in dementia and psychosis in Parkinson`s sickness (J. L. W. Bosboom, D. Stoffers, E. Ch. Wolters) •Growth/differentiation factor-15 (GDF-15), a unique member of the TGF-ß superfamily, promotes survival of lesioned mesencephalic dopaminergic neurons in vitro and in vivo and is triggered in neurons following cortical lesioning (J. Strelau, A. Schober, A. Sullivan, L. Schilling, ok. Unsicker) •Gene remedy for Parkinson`s sickness (H. Mochizuki, Y. Mizuno) •Stereotaxic intrastriatal implantation of human retinal pigment epithelial (hRPE) cells connected to gelatin microcarriers: a possible new phone remedy for Parkinson`s ailment (R. L. Watts, C. D. Raiser, N. P. Stover, M. L. Cornfeldt, A. W. Schweikert, R. C. Allen, T. Subramaniam, D. Doudet, C. R. Honey, R. A. E. Bakay)
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Additional resources for Advances in Research on Neurodegeneration: Volume 10
A new intramembrane integrative mechanism, Macmillan Press, London Fuxe K, Agnati LF (1991) Volume transmission in the brain. Novel mechanisms for neural transmission, Raven Press, New York Fuxe K, Fredholm B, Agnati LF, Corrodi H (1978) Dopamine receptors and ergot drugs. Evidence that an ergolene derivative is a differential agonist at subcortical limbic dopamine receptors. Brain Res 146: 295-311 Fuxe K, Agnati LF, Ogren S, Andersson K, Benfenati F (1983) Neurobiology of central monoamine neurotransmission: Functional neuroanatomy and noradrenaline and 5-hydroxytryptamine involvement in learning and memory.
In this setting, the term tolerance may be misleading, because mild autoimmune destruction may be a side effect of powerful benign autoimmunity. Self-tolerance in the immune privileged CNS 43 axonal degeneration lesion-independent danger signals destruction Fig. 7. How selective expression of B7-molecules may regulate tolerance versus autoimmunity. Axonal degeneration induces expression of B7-2 on microglia while data indicate that the development of CNS autoimmunity depends on the expression of B7 -Ion APCs.
Curr Opin Neurobioll0: 612-624 Andersson M, Hilbertson A, Cenci MA (1999) Striatal fosB expression is causally linked with L-DOPA-induced abnormal involuntary movements and the associated upregulation of striatal prodynorphin mRNA in a rat model of Parkinson's disease. Neurobiol Dis 6: 461--474 Angers S, Bouvier M (2000) Reply: beyond receptor dimerization. Trends Pharmacol Sci 21: 326 Bailey CH, Bartsch D, Kandel ER (1996) Toward a molecular definition of long-term memory storage. Proc Natl Acad Sci USA 93: 13445-13452 Bartlett F (1932) Remembering.